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To advance biogeochemical models, we seek to separate the effects of these storage zones on solute fate and cycling in streams.
With molecular models, we seek to understand how imprinted materials are created and what happens to TSAs and substrate molecules in these imprinted cavities to become eutomers.
In this regard, recent summit themes have ranged from "Shared Norms for a New Reality" (acknowledging that the 21st Century demands new normative behaviors, that which we should do) to "The Great Transformation" (allowing for the new models we seek for today's economic and governance realities).
Whilst it is challenging to bring historically-disparate partners together into formal agreements, they are essential to creating the scalable 'business rules' and sustainable environment required to achieve the new care models we seek.
In the present study, using both prostate cancer cell line and xenograft models, we seek to investigate the therapeutic potential of combining γ-T3 and PSP in the treatment of prostate cancer.
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Given the three distinctive subgroups of mouse HCC models, we sought to examine how well these models recapitulate human HCC phenotypes as defined by gene expression patterns.
Having demonstrated that apolipoprotein A-I (apoA-I) mimetic peptides ameliorate cancer in mouse models, we sought to determine the mechanism for the anti-tumorigenic function of these peptides.
Using difficult airway-simulated models, we sought to describe the time difference between placing the ETI and LT and the successful placement of each adjunct in varied levels of healthcare providers.
Because compensation occurs more likely in congenital loss-of-function models, we sought to investigate the effects of I/R in an inducible DKO mouse model.
As a consequence of the proposed difference in the mutational spectrum in CSC and differentiated cells of the two models, we sought to investigate this by mutational profiling of the different cancer cell populations.
As a first step in comparing these cell models, we sought to analyze the protein levels of insulin receptor β, IGF1R, AKT and ERK in a small initial pilot western blot study.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com