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Based on literature on human social models we hypothesized that the visual presence of the interface agent would result in more positive attitudes toward engineering and greater self-efficacy than the presence of a human voice alone.
On the basis of preclinical models, we hypothesized pharmacodynamic separation, achieved by intermittent delivery of epidermal growth factor receptor tyrosine kinase inhibitors intercalated with chemotherapy, as a reasonable strategy to deliver combination therapy.
Since we and other groups have reported that the inhibition of FAK activity exhibited a potent anticancer effect in several cancer models, we hypothesized that TAE226, a novel ATP-competitive tyrosine kinase inhibitor designed to target FAK, can prevent the occurrence and progression of peritoneal dissemination.
Moreover, consistent with GxE models we hypothesized that any genotype-related differences in brain function would be most pronounced in patients with PTSD.
Since misfolded tau has been shown to transmit across synapses in AD animal models, we hypothesized that synapses in AD patients may contain misfolded tau.
Given the influence of estrogen on SLE in experimental models, we hypothesized that OCPs with estrogenic effects would similarly accelerate lupus development.
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Unlike existing models, we hypothesize that the BG subserve the selection of both motor- and cognitive-related information in these tasks.
On the basis of animal models, we hypothesize herein that persons with metabolic syndrome have more seasonal changes in their mood and behavior.
Based on this model, we hypothesized that dyskinesias will develop predominantly in PD patients with a relatively preserved serotonergic system.
Using California as a model, we hypothesized that SNHs with trauma centers vary in terms of financial stability.
To test and validate this model we hypothesized that Mammary Serine Protease Inhibitor (maspin) acts as cytoskeleton regulator that mediates cell-extracellular matrix (ECM) adhesion in tumor.
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