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Although RS-predictor established different CYP450 isoforms prediction models, we cannot know in advance which isoforms of CYP450 will participate in the metabolic reactions.
Since the reduced forms of the popular measures of asymmetric information in the price formation process are not nested within larger models we cannot evaluate their fit using standard statistical tools.
Furthermore, employing non-conditional transgenic and mutant models we cannot rule out the possibility that artificial CD83 overexpression or deficiency on pro- and pre-B cells or stromal cells during B cell maturation within the CD83Tg and CD83mu mice results in the generation of dysfunctional B cells.
However, without such models we cannot hope to truly understand macrophages or indeed any other cell at a systems level.
14 15 In common with other predictors included in the models, we cannot be certain whether the observed relationship is causal or the result of Pakistani ethnicity being related to another factor or factors not otherwise captured in the models.
While we could adjust for these differences using multivariate regression models, we cannot exclude some degree of residual confounding or confounding by unmeasured factors such as a carryover effect of the previous anti-TNF agent.
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Due to the complexity of the model, we cannot check all properties at design time using Maude.
In the context of our model, we cannot assume that connectivity is either regular or bidirectionally symmetric.
However, with such a model, we cannot analyse the determinants of the efficiency levels in a single-stage procedure.
Nonetheless, it follows that in this model we cannot calculate the total impact of a rise of GDP in the total economy.
In this 3-D model, we cannot expect enough charge buildup at the ends of the elongated C1 conductor, which may lower apparent resistivity (Ting and Hohmann [1981]).
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