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It is such open and meritocratic models that developed and govern the internet and the world wide web.
Thus all tested mouse models that developed colitis showed bacteria in direct contact with the epithelium in the distal colon.
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The modelling outcomes categorized into models that develop with and without a subduction polarity reversal.
We address these issues through biologically-inspired computational models that develop representations through reinforcement learning.
Studies in mouse models that develop Aβ deposition have shown that murine apoE and human apoE4 have different abilities to facilitate plaque or CAA formation when studied independently.
There are also a number of genetically deficient mouse models that develop colitis [16].
Using this promoter, Connolly et al. have successfully developed the very first ovarian cancer transgenic models that develop ovarian carcinomas with metastatic spread to peritoneal organs [39].
Others have generated mouse models that develop tumors in the distal colon using implanted cancer cells [27] or adenovirus activated mutations [28].
Other genetic mouse models that develop multi-oocyte follicles have been reported.
Several fish models that develop the same disease as are seen in humans have been established.
In addition, there are spontaneous models that develop arthritis because of a single mutation.
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