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Immune-deficient animal models revealed an amelioration of cerebral ischemic-reperfusion injury [12], [18], [19].
Previous studies in various humanized mouse models revealed an impaired function of human B cells reflected by poor reconstitution of human IgM serum levels and limited ability to undergo immunoglobulin class switch and to secrete immunoglobulins other than IgM [15], [18], [20] [22].
Cyclin D knock out mice models revealed an intricate situation where different members of the family can partially compensate for each other depending on tissue expression and roles.
Multivariate adjusted regression models revealed an inverse association between maternal Hcy concentration and male birth weight (β = −210.40, standard error (SE) = 102.08, p = 0.04).
Combined line cross and half sib models revealed an additional drip loss pQTL on SSC4 in position S0214-S0097 (Liu et al., accepted).
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The models revealed a significant correlation between the predicted and observed creativity scores.
But, including SOB in factorial models revealed a previously unobserved association and evidence of moderation of DRD4 expression by SOB.
The immunological analysis in these two models revealed a relatively low level of tolerance against gp100, whilst T cell tolerance against gag was more profound.
All models revealed a stepwise increase in amputation risk with increasing triglyceride levels.
All models revealed a significantly increased OR for GDM for ethnic minority women.
The generated 3D models revealed a typically configuration of the sinonasal communication ways.
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