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Although thrombolytic agents such as streptokinase, urokinase and tPA have been proven to reverse the microvascular thrombosis in the animal models, reports suggesting their efficacy with human subjects are scanty (Askari et al. 2006).
In animal models, reports of progesterone's effects on bone density have been variable and are influenced by estrogen, the dose of progesterone administered, skeletal site analyzed, and the stage of skeletal maturation [19], [20].
This is consistent with previous animal models reports that TGF- β was increased in myopic sclera tissue [ 12, 29– 31] and also consistent with our previous study that documented that TGF- β could inhibit the growth of cultured human scleral fibroblasts [ 32].
Only three variables were significant in the crude models, reports of flood damage, which was unexpectedly associated with lower endotoxin, Hispanic subjects associated with higher endotoxin (in contrast to personal endotoxin), and high school or lower education level in mothers that was associated with lower endotoxin.
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This model was employed because it matches the modeling framework for the other models reported in this paper.
Late last month, Utah, the latest laboratory for Tsemberis's's models, reported it has nearly eradicated chronic homelessness.
The other three models report no discount rate.
All models report the marginal effects of logit regressions.
We test this contention with three analytical models reported in Table 1.
We do so by estimating ordered probit models (reported in the Appendix Tables 7 and 8).
All the models reported in the following are estimated by the "xtmixed" command in Stata13.0.
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