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The reason why designers still use physical models remains unclear.
Furthermore, the effect of HA addition on fibroblast function within in vitro models remains unclear.
However, the clinical relevance of these animal models remains unclear, and translation into clinical studies is difficult.
How exactly NAV3 aberrations support metastasis and tumor progression in patients and in animal models remains unclear.
Whether these traits are either necessary or sufficient for transmissibility among humans or even other mammalian animal models remains unclear.
However, whether exosomal tau release is a regulated neuronal process in vivo or results from overexpression and missorting of tau in these cell models remains unclear.
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Which model, remains unclear.
IL-22 and IL-17A were only upregulated after the peak mortality in the chicken embryo model occurred; thus, the role of the Th17 response in this model remains unclear.
The specific role of inflammation in this model remains unclear.
However, the impact of FGF10 on postnatal bone development and modeling remains unclear.
The role of the other 12 proteases and antiproteases which are over-expressed in our model remains unclear.
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