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This activation mechanism differs fundamentally from the intracomplex initiation models previously proposed for both the lectin and the classical pathway.
Our results evidence the not negligible contribution of erosion and associated flexural rebound to the uplift of the Cordillera Blanca and allow us to question the models previously proposed for the formation of the CBNF.
The low noise but highly plastic expression is therefore consistent with two (not mutually exclusive) models previously proposed.
Such physical separation rules out the majority of models previously proposed for the mechanism of complex I, based on the assumption of direct coupling between the chemistry of Q reduction and the vectorial movement of protons across the membrane.
Nevertheless, the multigenic duplication model for the mouse H2-T region is similar to the multigenic tandem duplication models previously proposed for the Mhc class I region of human and non-human primates [ 28, 29].
This mechanism differs from the pathway models previously proposed for endothelial cells and other types of neurons, which were derived exclusively from in vitro experiments (Fig. 7F,G; see Discussion).
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The results were interpreted in terms of a physically-based constitutive model framework previously proposed.
The work provides an upgrade of the model previously proposed by the author [1].
The present researcher's conceptual model is based on a model previously proposed by Keramati and Ardabili (2011).
The model represents an enhanced version and a generalization of another model previously proposed by the authors.
The model is also used to validate a steady-state design model previously proposed by Esposito et al. [1].
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