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In this concern, we can speculate that decreased effective connectivity from the ITG to the amygdala might be the neurobiological correlate of the altered perceptive activity known as attentional bias in the cognitive models of SAD.
These data, along with our findings, supported the notion that DMN accounts for prominence in cognitive behavioral models of SAD.
As a more objective approach, neuroimaging holds great promise for detecting abnormalities crucial to the pathophysiologic models of SAD.
They argue, instead, these changes primarily improve consistency between diagnostic criteria and common theoretical and clinical models of SAD.
Cognitive models of SAD (Clark 2005; Hofmann 2007; Rapee and Heimberg 1997) argue that a biased perception of how others view them is central to SAD.
Indeed, current cognitive models of SAD state that safety behaviors play an essential role in the maintenance of SAD (Clark 2001; Hofmann 2007; Rapee and Heimberg 1997).
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Christianne Stotijn's Brangaene was a little colourless, and too often wordless also, Matthew Best's King Marke was a model of sad dignity.
One method to evaluate the relative performance of the models would be Bayesian Model Selection (Penny et al. 2004), which could also be used to compare the model structures of the winning models of the SAD and HC groups.
To date, there is no clear neuroanatomical model for SAD; however, an increasing amount of the neuroscience literature is being devoted to social functioning.
Ji et al. [2] refined Sousa's model using a combination of SAD and row-based SAD as the threshold criterion.
CT was based on Clark and Wells' (1995) model of the maintenance of SAD and followed a detailed German manual (Stangier, Clark, & Ehlers, 2006).
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