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Hiratani, I. et al. Genome-wide dynamics of replication timing revealed by in vitro models of mouse embryogenesis.
Furthermore, an absence of JNK1 results in decreased adiposity, significantly improved insulin sensitivity and enhanced insulin receptor signalling capacity in two different models of mouse obesity.
The Mouse Brain Web (MBW), a web-organized database, provides for the construction of anatomically correct models of mouse brain networks.
Calculations were performed for the models of mouse, rat and man.
Other researchers like Parsons and Sera et al. have utilized IL-XPCI to study lungs, and successfully generated the three-dimensional models of mouse airways [7], [17].
Using models of mouse embryonic fibroblasts (MEF) and human cancer cells lines, we were able to show that the PINCH1/PP1α/AKT1 interrelation is responsible for increased cellular resistance to both X-ray irradiation and the chemotherapeutics cisplatin, 5-fluorouracil and gemcitabine [23].
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In the present study, even though ALA-treatment might promote eating behavior and thus gain body weight, the blood glucose levels reduced in a wide spectrum of disease models of mice, including obesity, aging and chronic kidney disease.
There are genetic models of mice that have abnormalities with drinking.
We went further than our previous study by including data from a number of different models of mouse-adapted scrapie.
Gottfries, J., Melgar, S. & Michaëlsson, E. Modelling of mouse experimental colitis by global property screens: a holistic approach to assess drug effects in inflammatory bowel disease.
This may explain the reason why anti-Forssman antibody per se fails to induce hyperacute rejection in a xenotransplantation model of mouse heart xenografts to rat5.
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