Sentence examples for models of dilated from inspiring English sources

We performed an initial evaluation of non-invasive ECG-gated [18 F]FDG-positron emission tomography (FDG-PET) for serial measurements of left ventricular volumes and function in murine models of dilated (DCM) and ischemic cardiomyopathy (ICM), and then tested the effect of erythropoietin (EPO) treatment on DCM mice in a preliminary FDG-PET therapy monitoring study.

Relative message abundance was also determined in GLUT4 null mice and in murine models of dilated and hypertrophic cardiomyopathy.

For example, GLP -1 has been found to exert cardioprotective actions in experimental models of dilated cardiomyopathy, hypertensive heart failure, and myocardial infarction.

GLP-1 also protects against LV dysfunction in experimental models of dilated cardiomyopathy, hypertensive CHF and MI, at least partly by increasing myocardial glucose uptake [ 14, 16, 21].

Several processes are responsible for these adverse changes including impaired calcium cycling, myocardial insulin resistance, increased lipid uptake, glucotoxicity, and activation of the renin-angiotensin-aldosterone system observed in mouse models of dilated cardiomyopathy (Table 2) (20).

Therefore, a potential application of our construct would be the generation of clinically relevant, large animal models of dilated cardiomyopathy, which are currently unavailable and urgently required to study pathophysiological alterations and test new therapeutic interventions [ 26].

Finally, in vivo tests of the biocompatibility of the fabricated scaffolds have been done using a mouse model of dilated cardiomyopathy.

We now interrogate whether cardiomyocyte-specific, transgenic CRNK expression prevents LV remodeling and HF in a mouse model of dilated cardiomyopathy (DCM) caused by constitutively active Protein Kinase Cε (caPKCε).

We report here the beneficial effects of the GLP-1 agonist exenatide, which can be administered by intermittent subcutaneous injection, on glucose homeostasis, cardiac function and survival in a transgenic mouse model of dilated cardiomyopathy.

Importantly, transplantation of MSCs led to decreased fibrosis in a rat model of dilated cardiomyopathy [ 60].

The dynamics of these modules between normal and disease states further suggest a potential molecular model of dilated cardiomyopathy.