Sentence examples for models of collagen from inspiring English sources

Telfer, W. R., Busta, A. S., Bonnemann, C. G., Feldman, E. L. & Dowling, J. J. Zebrafish models of collagen VI-related myopathies.

The smallest units of these fibrillar structures have cross-sections of ∼3 × 5 nm, consistent with current models of collagen microfibril formation.

Next to these novel insights, the unique human data set created in this study provides valuable input for the development of numerical models of collagen remodeling and optimization of tissue engineering.

H4R-deficient mice and wild-type mice administered the H4R antagonist were studied in models of collagen antibody-induced arthritis (CAIA) and collagen-induced arthritis (CIA).

Most previous models of collagen proteolysis assumed that the fibrillar arrangement would freely accommodate MMPs to digest individual collagen monomers in the fibril [112,113].

Anti-apoptotic strategies, such as blocking the mitochondrial permeability transition pore by treatment with cyclosporine A, have been shown to rescue mouse models of collagen VI deficiency (22) and correct mitochondrial dysfunction and muscle apoptosis in patients with collagen VI myopathies (19).

We and others have demonstrated previously that bone marrow derived DC genetically modified to express IL-4 reduce disease pathology in mouse models of collagen-induced arthritis and delayed-type hypersensitivity [19], [20].

We and others have previously demonstrated that treatment with bone marrow derived DC genetically modified to express IL-4 reduce disease pathology in mouse models of collagen-induced arthritis and delayed-type hypersensitivity.

The animal models of collagen-induced arthritis reported varied results.

Studies using animal models of collagen-induced arthritis and IFN- α-induced lupus nephritis actually already provide prove of concept.

IL-17A appears to be associated with chronicity as demonstrated by murine models of collagen-induced arthritis.