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Instead of devising experiments and carrying them out directly, Carl builds computer models of bacterial populations and investigates them virtually.
These are important considerations for distinguishing between various models of bacterial migration for field-scale applications.
Here we present the use of genetically tractable Drosophila models of bacterial pathogenesis to study infection with the human opportunistic pathogen Pseudomonas aeruginosa.
This approach reduced vascular permeability in the lung and other organs and increased survival in animal models of bacterial endotoxin exposure, polymicrobial sepsis, and H5N1 influenza.
Many animal models of bacterial diseases are hampered by differences in tissue tropism and the course of pathogenesis.
We compare models of bacterial and eukaryotic condensins in order to uncover conserved mechanistic principles of condensin action and to propose a model for mitotic chromosome condensation.
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Nehme, N.T. et al. A model of bacterial intestinal infections in Drosophila melanogaster.
An in vitro model of bacterial infections in wounds and other soft tissues.
Nguyen, T. T. et al. Mathematical modeling of bacterial kinetics to predict the impact of antibiotic colonic exposure and treatment duration on the amount of resistant enterobacteria excreted.
We quantify homologous recombination in five bacterial pathogens and use these parameter estimates in a multilocus model of bacterial evolution with additive and epistatic effects.
A prototype kinetic model of bacterial glycolysis is formulated and a minimal set of assumptions is postulated that allows the formulation of an approximate mathematical model.
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Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com