No single animal model of arthritis truly represents the human disease, but the models mimic various aspects and can be used as tools to understand particular pathways.
These models mimic various human leukemogenic processes in the context of distinct disease genotypes and phenotypes associated with the different forms of AML [ 20].
These models are robust in mimicking various aspects of cell population, but fail to examine the effects of cell deformation and morphology on pattern formation and growth processes.
It was shown that these cell models can mimic various topologies of tissue such as cyst or tumor or monolayer.
Gain and loss of function studies in various animal models mimicking human disease have shed light on the functional role and target specification of microRNAs.
Considering the major pathways involved in the etiology of cataract such as polyol pathway or sorbitol accumulation pathway, anticataract potential of PHF was evaluated using various pharmacological screening models mimicking the major pathways in cataract.
In vitro models span from simple aqueous solutions that mimic the lung surface epithelial lining fluid, to cell culture models, to tissue explants and slices, to intact isolated lungs, and finally to artificial models which attempt to mimic various aspects of the tracheobronchial tree.
To better illustrate the dependence of transient times on these two properties, we introduce a simple model oscillator that can mimic various oscillators.
A major limitation in the development of an appropriate drug and delivery system for acne is the unavailability of a suitable animal model that can certainly mimic various pathophysiologic characteristics in humans.
Fetal sheep model is widely used to mimic various fetal conditions, yet ECG recordings in a public data set form are not available to test the predictive ability of fECG and FHR.
