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Cervical spinal cord injury (SCI) models in rats have become increasingly useful because of their translational potential.
These data, however, originate from un-resuscitated, short-term models in rats, whereas we studied long-term, fully resuscitated porcine septic shock.
Therapeutic angiogenesis was induced by local autologous bone marrow cell implantation (BMCI) in ischemic hindlimb or ischemic heart models in rats.
Open image in new window Fig. 1 Effect of graded dose of xyloccensins X+Y and reference drug omeprazole (Omz) on percentage protection of ulcer against cold restraint induced gastric ulcer models in rats.
Several groups have also evaluated neuroinflammation models in rats using the TSPO radioligand 18 F-DPA-714, concluding that it provides accurate quantitative information of TSPO density after cerebral ischemia, herpes encephalitis, amyotrophic lateral sclerosis, and gliomas [24 27].
n = 6 in each group Open image in new window Fig. 2 Effect of xyloccensins X+Y and reference drug sucralfate (SUC) on percentage protection of ulcer against alcohol induced gastric ulcer models in rats.
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These data suggest the successful establishment of a neuropathic pain model in rats.
The comparative study was performed using the BTX injection model in rats (n = 5 for each group).
In studies 1 and 2, we used a syngeneic tumor model in rats with an uncompromised immune system.
Our findings demonstrated that HBO treatment significantly suppressed mechanical and thermal hypersensitivity in the CCI neuropathic pain model in rats.
Gastric damage was evaluated in the absolute ethanol-induced ulcer model in rats.
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