Sentence examples for models in panel from inspiring English sources

Models in Panel a and Panel b are otherwise identical; other controls are shown at the bottom of the table.

The relative head domain orientations of the II-c and II-e subclass density maps are indicated as cartoon models in panel (E) and (G), respectively.

Models in Panel A include separate controls for short and long term bank debt as a share of the firms turn over, the log of the firm's turn over, the log of workers and indicators for zero values of sales and debt.

A key difference between the electrophilic activation model shown in panel A and the alternative models in panels B and C is that in the electrophilic activation model, the disulfide bond is labilized (i.e., activated for attack), whereas models B and C only stabilize the product of the reaction.

Models in panels E and F of Figure 4 show insertion of the antiparallel β-sheet into a single leaflet rather than membrane traversal analogous to that of bacterial porins.

b Cross-sectional topographic profiles of the basin, extracted from the MOLA topography model in panel a. c Geology map of the study area.

Furthermore, referring to the final model in panel A, several of our control variables (MV, IFF as well as the index dummies) are significant and show the predicted signs, which indicates that characteristics of a firm's information environment are relevant for the width of bid ask spreads.

Indeed, our model reveals that system optimization of individual myelinated axons is achieved when the inner diameter approaches 77% of the outer diameter, i.e. a g-ratio of 0.76 0.77 (Figure 3A D; theoretical model in panel B: r = 1.0, R2 = 0.99, p<0.0001; model fit to experimental data in panel D: R2>0.96 for each).

We felt that the old model in panel A was needed to contrast with the new model in panel B. We also felt that panels C and D were needed to explain the changes that occur with LTP (panel C) and LTD (panel D).

The inverse nature of these associations by genotype, from the blood lead model in panel 2 of Table 3, is shown graphically in Figure 1.

Structures in panels A – C are aligned with the no-nucleotide K349 model in panel D by strand b7 of the central beta sheet, which connects to the switch II loop; this alignment approximately maintains the orientation of the upper subdomain, thus preserving a contact between L8 within this subdomain and alpha tubulin (see Figure 2 ).