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Co-evolutionary models hypothesize that different social structures arise from the same evolved individual psychological mechanisms responding to different environmental cues.

Both the rigid- and flexible-rotor models hypothesize that the protons enter the Fo complex via a channel buried in the rotor-stator interface from the 'outer' space halfway into the membrane, and later leave the complex from a putative channel located inside the cytosolic/inner half of the stator (a-subunit) (Fig. 1).

Although current models hypothesize that immune cells promote vascular permeability in human disease, the role CD8 T cells play in BBB breakdown remains poorly defined.

The third model postulates that abiotic factors structure communities, and these environmental models hypothesize that species distributions are related to environmental conditions [39], [40] and/or sources of human impact [41], [42].

Although current models hypothesize that immune cells promote opening of the BBB in human disease, the role of CNS infiltrating antigen specific CD8 T cells in initiating vascular permeability has not been determined.

The clinical staging models hypothesize that there are stages (0, 1a and 1b) that precede full-blown BP.

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The cross-lagged model hypothesizes that agency at t1 has a positive effect on agency at t2 (H1) and that outcome at t1 has a positive effect on outcome at t2 (H2).

A new disease model hypothesizes that thyroid hormone receptor-associated protein 3 (Thrap3) could activate a diabetogenic gene cascade in adipose cells through interaction with cyclin-dependent kinase 5 (CDK5) leading subsequently to the phosphorylation of peroxisome proliferator-activated receptor γ (PPARγ) at Ser273 (Choi et al. 2014).

The model hypothesized that plugging occurred via deep-bed filtration and incorporated physical effects of porosity and effective specific surface area changes due to the capture of fines, inertial effects of phases, and coupling effects between the fines filter rate equation and the interfacial momentum exchange force terms.

The model hypothesizes that number meaning is represented by differentially tuned neuronal populations giving maximal response to their preferred numerosity.

Our model hypothesizes that point mutations, which altered base compositions (i.e., GC-to-AT or AT-to-GC), took place before the occurrence of gBGC.

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