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All models have presented with significant validity problems relevant to a hypothesized etiological stress model of depression.
Second, previous models have presented few mechanisms for accurately quantifying the infection-induced inflammation, which is responsible for the clinical symptoms of swelling, immobility and pain.
Several commentaries, cost-effectiveness studies and models have presented potential uses for and likely impacts of this assay [ 14- 21].
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In the case of Group II the PLS models (FTIR-ATR and FTNIR) have shown the same accuracies, while the ANN/FTNIR models has presented better performance than the ANN/FTIR-ATR models.
The model has presented a suitable probability of good classification for the external prediction data set.
In addition, mathematical modeling has presented important concepts about CRISPR dynamics during phage-bacteria interactions [ 18, 19].
In this section, we will describe the implementation of the group-analysis model, having presented the theory behind this model in the last section.
In our models, we have presented the vector-host model with their stability analysis.
Moreover, the models we have presented assume identically and independently distributed genotypes.
The models we have presented have the considerable advantage of being able to handle several factors simultaneously, but the choice of which to incorporate lies with the analyst.
Early in the book, he claims that "the model I have presented is the only possible model that satisfies all the constraints that the research and our thought experiments have established".
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CEO of Professional Science Editing for Scientists @ prosciediting.com