Yet, to date, no computational model has explicitly addressed the mechanisms that give rise to both goal-specific and goal-independent parietal mirror neurons.
We conclude that our VZV individual-based model has explicitly estimated the duration of exogenous boosting to be limited to only 1 or 2 years and that there was no significant effect from endogenous boosting.
Some of the most compelling evidence supporting the view that particular pools of Aβ can be especially pathogenic comes from comparison of the relatively few animal models that have explicitly directed Aβ exclusively to one compartment.
Though multiple previous modeling studies have explicitly included protein DNA interactions (e.g., in Drosophila, see He et al., 2010; Janssens et al., 2006; Junion et al., 2012; Kazemian et al., 2010; Segal et al., 2008; Zinzen et al., 2009), here, we choose to model the relationship between inputs and outputs directly as this offers several advantages.
Although other models have not explicitly controlled spheroid shape, they have nevertheless done so implicitly.
Second, although the literature recognises that individual's PA behaviour is unstable, the economic models have not explicitly accounted for this, but rather assumed constancy of behaviour once people settle in the active state.
However, previous theoretical models have not explicitly addressed how females' own heterozygosity might influence their mating preferences, and have overlooked the fact that only homozygous females can increase the heterozygosity of their offspring by mating with heterozygous males (e.g., see Table 2 in [ 12]).
However, being content with establishing population-level trends, few studies have explicitly modeled the source of this variation.
Few studies have explicitly modeled the potential relationships between climate change, air quality, and human health, and fewer still have investigated the sensitivity of estimates to the underlying modeling choices.
