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The authors of the models have evaluated concentration changes of major apoptotic molecules by Western blot analysis and represented them as relative values at given time points.
A summary of the issues involved in modelling HIVST is provided in Table 1.> Several mathematical models have evaluated the impact and/or cost-effectiveness of expanding HTC in some high income [ 20– 25] as well as low and middle income countries [ 26– 29].
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Other studies using a similar non-insulin DM model have evaluated cardiac function, however at later time points, such as 2 and half months and 6 months following STZ injection [ 31, 32].
The energy model has evaluated the proposed energy conservation measures and the best practices that were generated by the energy management program steps.
Further modeling has evaluated larger, neural circuitry models (column and local circuit scales) made up of thousands of biophysically detailed multicompartmental neurons (ion channel, dendrite and cell scales) with synaptic dynamics.
Rodent studies reviewed below have analyzed a variety of fibrosis progression and resolution models, and have evaluated liver tissue, cell isolates, serum and urine proteomes.
Using this model, we have evaluated cephalosporins, aminoglycosides, fluoro-quinolones, and carbapenemes in combination with G-CSF.
Using this cell line model, we have evaluated the BCAC hypothesis that low-risk SNPs confer breast cancer risks by modulation of expression levels of nearby located genes.
Several studies in model plants have evaluated the importance of this hormone in crosstalk signaling with different metabolic pathways, in addition to responses to biotic stresses.
For example, we have evaluated models in which connections are probabilistic.
Despite convincing evidence in animal models, few epidemiologic studies have evaluated PON1 status as a determinant of OP sensitivity.
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