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Classical models have attributed the radiosensitization to the physical dose enhancement arising from the keV electrons.
Studies in both human patients and the animal models have attributed the pathogenesis of TB IRIS to hyperactive IFNγ+CD4+ T cell responses [3, 5].
Population genetic models have attributed 39% [ 2] and 18%38%% [ 1] of human infection in the United Kingdom to ruminant (cattle or sheep) sources, and 20%30%% in New Zealand [ 3], with cattle identified as the main ruminant source.
The neural generators of Ta are not well defined at present, but dipole models have attributed Tb activity to bilateral radial dipoles in the temporal lobe (Scherg and Von Cramon, 1985 , 1986 Ponton et al., 2002).
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Other animal model studies have attributed toxicity of cephalosporins to cortical AUC (area under the curve), direct effect of the lactam ring and disruption of the mitochondrial respiratory enzymes in the cortical tubular cells [ 13, 14].
Although previous CA models for aeolian dunes have attributed the bedform generation to changes in the saltation length, in the present study, which employs a stochastic model, we consider changes of the migration speed of sand particles that are in continuous contact with the bed (i.e., sand that moves no faster than one cell per time step).
Some analysts have attributed these deficiencies to weaknesses in the integration model's design and implementation [ 10- 12].
Some have attributed the accident to suicide.
Modelling work has attributed only a minority of the decline in HIV-2 from its peak to competition with HIV-1 [ 17].
Further complicating the interpretation of the data is that differentiation events in animal models have been attributed to differentiation, transdifferentiation, cell fusion or surface antigen transfer (trogocytosis), depending on the donor cells and the animal model being used.
Skeletal muscle is responsible for 30 40% of resting metabolic rate (Zurlo et al, 1990), and consequently, lean phenotypes of many mouse models have been attributed to enhanced muscle metabolism (Reitman, 2002; Gilliam & Neufer, 2012).
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