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Ongoing bone growth should be considered when interpreting reports that use animal models for bone and joint diseases.
Mouse models for bone marrow transplantation with hematopoietic stem cells (HSC) have become the paradigm for stem cells analysis since their discovery almost 50 years ago [1], [2].
Validation of these models for bone lead release has been limited, however, because of the scarcity of suitable pediatric data for accurate ground-truthing and calibration.
Towards this end, in vivo studies of alanine-modified polyphosphazenes on rat and rabbit models for bone tissue engineering materials have been reported [ 5, 47].
Advances in the field of mechanobiology in bone repair could be enhanced through a common consensus for standard animal models for bone healing, fixation techniques, outcome measures and standard comparison metrics.
All 189 patients were included in several analysis of covariance models for bone mineral density (BMD) variations (final values minus initial values) to analyze the effect of cofactors (continuous variables and discrete variables) on that of infliximab.
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Aiming at overcoming the limitations of the plaster-casting method in traditional prosthetic socket fabrication, the idea of reconstructing the 3D models for bones and skin of the residual limb is proposed.
The model for bone injury under disuse status is established.
This work presents a computational model for bone remodelling around cementless stems.
In this work, a three-dimensional model for bone remodeling is presented, taking into account the hierarchical structure of bone.
We present a multiscale model for bone tissue adaptation that considers the two top levels, whole bone and trabecular architecture.
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