Both models demonstrated similar model performance.
Despite different levels of simplification of the phenomena, simulations using both models demonstrated similar mechanisms, including the progress of a sharp thawing front separating a frozen, non-impregnated inner layer from a thawed, highly impregnated outer (surface) layer.
Both CART and risk score models demonstrated similar excellent predictive abilities (81.9% and 84.5%, resp).
Finally, practical applicability and model simplicity were considered if the models demonstrated similar MAE and R values.
The two disease models demonstrated similar behavioral deficits, such as hyperactivity, poor working memory performance, and social withdrawal.
These additional models demonstrated similar results to those reported here.> -wrap-foot> a RF negative was used as a reference group; badjusted for age at symptom onset, sex, baseline smoking status, year of inclusion in cohort and inflammatory marker; cACPA negative was used as a reference group.
While no animal allergy model is completely representative of human allergy, probiotic modulation of immune responses in this pig allergy model demonstrated similar probiotic modulation of immune responses as was seen in human clinical trials, and may predict probiotic modulation of allergic lung responses in humans.
In addition, the authors show that their SCMGENE model demonstrated similar prognostic implications when compared to more established models such as PAM50, MAMMAPRINT, and ONCOTYPE gene expression signatures, and may be adequate for clinical management of patients.
Stratifying this model demonstrated similar associations with comorbidity, whether or not peptic ulcer coding was present, and slightly higher associations for a peptic ulcer with exposure to previous peptic ulcers, NSAID, or aspirin use (Table 3).
As summarised in Table 2, these models demonstrate many similar features but also have interesting differences that impact our understanding of haematopoiesis and MPNs.
