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To establish a link between mathematical models and biological systems an approach based on process algebra is used for agents based communication (Sumpter and Blanchard 2001).
In another research, an approach based on process algebra is used for agents based communication to establish a link between mathematical models and biological systems (Sumpter and Blanchard 2001).
This review describes the discovery of XMRV, and the molecular and virological events leading to its formation, XMRV infection in animal models and biological effects on infected cells.
We provide a method for assessing the statistical significance of the fit between random graph models and biological networks based on non-parametric tests.
In addition to characterization in in vitro models and biological models with pharmacological and genetic manipulation of metal content, we have introduced the concept of metal-unresponsive control dyes and their testing alongside metal-responsive dyes to establish when metal-responsive dyes report on changes in labile metal pools.
Thinking in lines of the network models and (biological) theories of motivational orientation (e.g. Strack and Deutsch 2004; Davidson 1992 , 1993, one could also argue that sad mood would facilitate avoidance tendency, while positive mood would be characterized by a general approach tendency, regardless of the valence of the reference object.
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See [1] for the derivation of this model and biological justifications for the system parameters.
The synthesis, molecular modeling and biological evaluation of substituted deoxybenzoins and optimized dihydrostilbenes are reported.
Such information is directly relevant to the existence and properties of lipid raft structures in both model and biological bilayers.
As discussed above, one major advantage of disease modeling and biological screens based on iPSCs is the availability of patient-specific cells with defined genetic background that theoretically allows the direct correlation of the observed cellular phenotype with clinical data.
Combining computational modeling and biological data, we propose that the CCM protein complex functions in the PI3K signaling pathway through the interaction between PDCD10 and PtdIns 3,4,5 P3.
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