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The developed models also displayed high performance while validated through independent data sets.
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We find that the internally generated heating in WACCM3 and eddy dissipation values are both smaller than the values specified in the GSWM; the eddy dissipation fields and zonal mean zonal winds of the two models also display measurable differences in spatial structure.
Cancer models also display evidence of context-specific sulfatase activity.
Some models also display poor regulation of locus coeruleus neurons and noradrenergic hyperactivity.
All values are within an acceptable range and the modified models also display satisfactory local values.
Diabetic mouse models also display an increase in myocardial oxygen consumption.
However, although other CDG models also display evidence of overelaborated motor neuron architectures (Cline et al., 2012), the correlation with loss of HRP epitopes is less clear.
Various NS mice and zebrafish models also display general growth defects, such as reduced weight and short stature (Araki et al., 2004; Hernández-Porras et al., 2014; Jopling et al., 2007).
Starting from the same chromosome positions, the Type II model also displayed a tendency for chromosomes to individualize.
The variance analysis of regression model also displayed that X1, X2 were significant, and the interaction term X1 X3, a square term X1, demonstrated high significance.
Further, this animal model also displayed developmental defects, likely due to NCC migration defects, such as facial dysmorphology consisting of short and malformed palatoquadrate and Meckel's cartilage, components of the mandibular arch, and missing or smaller pharyngeal arches.
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