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The remaining models also demonstrate high accuracy, with at least 90% of the full training set (>2,546 systems) predicted to within 25% of the calculated values.
But the two models also demonstrate a key disagreement about what sustainability will look like in the future: will it be high-tech with an EV in the garage, or low-tech and tiny?
(69) Status epilepticus models also demonstrate a varying clinical response of the animal subjects to differing doses of convulsive agents.
The electrical kindling models also demonstrate seizures with progressive increase in frequency and severity following prolonged stimulation.
Previous torsinA loss-of-function models also demonstrate cell loss restricted to a neurodevelopmental window (Liang et al., 2014).
Consistently, recent studies on animal models also demonstrate a role of the Hippo pathway and YAP in tissue regeneration [ 18, 19].
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Characterization of these models also demonstrated increased proteolysis of collagen IV in the matrix by the PN driver cells as compared to wild-type SCs.
Evidence from murine models also demonstrates a central role in islet proliferation for both CDKN2A and CDKN2B[ 29, 29].
Moreover, studies in genetic or diet induced hypercholesterolemic mouse models also demonstrated a clear association between high lipid levels and BC development [ 21] and progression [ 22, 23].
Several mouse knockout models also demonstrated a significant role of the SCN in maintenance of glucose homeostasis– Clock mutant mice are hyperphagic, obese, and hyperglycemic.
The use of probiotics, particularly in animal models, also demonstrates that their secreted products or metabolites can modulate contractility of intestinal smooth muscle and visceral sensitivity.
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