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"By using both biochemical analysis and mathematical modelling we demonstrated how the core circadian clock keeps to a 24-hour cycle despite temperature changes and metabolic changes.
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By using the DSS-induced colitis mouse model, we demonstrated that miR-223 deficiency resulted in more severe symptoms.
By checking the differences between the simulated and recovered models, we demonstrated the reliability of our method.
By applying the model, we demonstrated 83.9% Cu, 96.0% Cr, and 99.3% As removal from treated wood particles.
By using the model, we demonstrated the neuronal growth process from disconnected neurons to fully connected three-dimensional networks.
By nuclear magnetic resonance and molecular modeling, we demonstrated that the YSNSG cyclopeptide actually adopted the expected β-turn conformation.
Using the 6-OHDA model, we demonstrated that SVs derived from LPS-stimulated primary astrocytes significantly accelerated DA neuron loss following 6-OHDA treatment.
In a mouse burn model, we demonstrated differential GR levels, size, and transcriptional activity in CD14 knockout (KO) mice when compared to wild-type (C57BL/6J) after injury.
Using a plasmid-based transient expression model, we demonstrated that N796, N580 and N799 were capable of significantly inhibiting viral replication in vitro and in vivo.
Using a rabbit model, we demonstrated that mechanical loading applied to cancellous bone in situ increased bone formation and altered trabecular morphology.
Using a knock-out mouse model, we demonstrated that the in vivo glucose metabolism can be affected by alterations in the zinc transporter system.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com