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Recent numerical modelling studies demonstrated how pre-existing (geologically older) fault geometries within a rock volume, strongly control both the distribution of strain and fluid flow patterns during extensional fault reactivation.
Modelling studies demonstrated that monomeric α-syn can rapidly penetrate the lipid bilayer and incorporate additional monomers to form pore-like structures that fully perforate the lipid bilayer [ 49].
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On the other hand, modeling studies demonstrate that there is most likely substantial current dispersion under the electrodes, especially at the cerebrospinal fluid level, where the conductance is particularly high [34], [53].
Neural network modeling studies demonstrate that scale-invariant contour grouping is indeed possible in such a hierarchical network (Korjoukov & Roelfsema, manuscript in preparation).
3 5 Economic modelling studies demonstrate how early interventions and investment in disadvantaged children are effective in improving child development and cost-effective in improving the 'human capital' potential of individuals.
The molecular modeling studies demonstrated that compounds matched the pharmacophore well and had good predicted physicochemical properties and estimated IC50 values.
Previous mouse model studies demonstrated immunization with r-GV expressing selected exogenous sequences elicited a prolonged immune response.
Previous DS animal model studies demonstrated that ETS2 had a tumor suppression role in intestinal tumor oncogenesis (41).
Several works in epidemiology and animal model studies demonstrated that compounds which possess antioxidant or anti-inflammatory properties can inhibit carcinogenesis [ 6, 7, 8, 9].
Animal model studies demonstrated that loss of TACSTD2 did not alter the incidence and natural history of papilloma formation, but enhance malignant phenotype of skin cancers through promoting EMT.
We review evidence from clinical data sets and mouse model studies demonstrating enhanced inflammation and altered tumor microenvironment (TME) upon TS inactivation.
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