Sentence examples for modelling led us from inspiring English sources

A noted ceiling effect on postoperative Oxford scores (ie, potentially problematic when performing regression modelling) led us to review our data.

The desire to maximize the size of the validation set to minimize the effects of noise in assessing the performance of the model led us to use an unusually small fraction of the available data to train the models – 10 to 30% instead of the typical 80 to 90%.

The use of this IV model led us to identify, among several factors investigated, the effect of aging and that of inosine as significant in reducing inflammatory reactions, and therefore improving P. falciparum growth.

Showing that inhibition or knockdown of the receptor leads to the functional results observed in the cellular assay model, led us to analyze combinatorial effects of using RNAi and kinase inhibitors in tandem.

The large uncertainty with respect to duct modeling led us to adopt a worst-case approach and use the spore disengagement rate that maximizes the number of anthrax cases.

This whole work, both the literature study and the establishment of various models led us to estimate different assumptions on the relationship between health and income.

Results from site-directed mutagenesis, hydropathy analysis, and homology modeling led us to identify a putative membrane association segment near the end of helix F in TXAS.

Deterministic and stochastic models led us to focus on basal transcription to optimize circuit performance and helped to explain the resulting circuit robustness across a large range of component expression levels.

Numerical thermal modeling led us to identify two thermal contributions to the interface recoil that increase with the pulling rate and add to the classical recoil associated with the solute boundary layer formation.

This unexpected change in lung mechanical properties in a healthy animal model led us to consider the possibility that functional residual capacity had increased in prone position and to look for lung recruitment as a possible explanation for this increase in lung gas content.

This model led us to observe and compare concomitant pathophysiologic pathways.