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The mono-allelic underdominance modeled here describes the situation where there is a transgenic allele at a single autosomal locus (the site of the transgenic construct integration).
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Thus, the experimental model here described may offer new opportunities to investigate the mechanisms implicated in placental malaria and in pregnancy outcome impairments, which are difficult to study in human subjects.
Together, the model here described should permit to further evaluate the significance and prognostic impact of the shift of micrometastatic cells from a cluster-aggregated into a single-cell status.
To extract seasonality patterns, a simple additive decomposition was performed with the following model (here described for ν, but a similar equation was used for π): (1) where, ν t represents the estimated quarterly values for ν, such that for Q quarters { ν t : t = 1,2,…, Q} = { ν1, ν2,…, ν Q }.
The model presented here describes the failure of those beams.
The model presented here describes the evolution of the damage up to failure of the first ply.
The model presented here describes in mathematical terms the action of the immune system on muscle tissues subsequent to damage triggered by a not well-specified mechanism.
The importance of this extension in VB modeling is here described and underlined.
The restriction to interval (0,1) was necessary as the model used here (described in Section Evaluation criteria) requires positive expression values for all genes, and facilitates computation by restricting its output to the same interval.
The O-PLS models are here described with the statistical parameters explained by variation (R) and predicted variation (Q).
The work presented here describes the model that has been used to design these devices.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com