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A regression model with gestational age at delivery as the dependent variable was constructed and repeated with neonatal morbidity as the dependent variable.
Introducing BMI or weight, but not AFA or AMA, in a model with gestational age, reduced the association between AEE and gestational age.
Linear regression revealed that smaller thalamic volume was associated with increased mean thalamic diffusivity when entered into a model with gestational age at birth, total brain volume, and cortical volume (Fig. 8; partial r = −0.395, P = 0.001).
Presence of a hemodynamically significant PDA prolongs the time to full feeds by 8.0 ± 2.8 days in the multivariable model with CRIB II score and by 6.8 ± 3.0 days in the model with gestational age.
Within these regions, linear regression showed that decreasing thalamic volume was independently associated with increasing radial diffusivity (partial r = −0.34, P = 0.004) but not with axial diffusivity (partial r = 0.008) when entered into a model with gestational age at birth and age at scan.
Similar(55)
The models with gestational age/preterm birth as outcomes were adjusted for maternal covariables including age, BMI, parity, ethnicity, smoking status, the presence of gestational diabetes or pre-eclampsia, blood pressure at booking and 36 weeks gestation, and IMD score.
Multivariate models with gestational age, type of delivery, Apgar score at 1st minute, and the presence of at least 0.0010% HSCs in CB as independent variables showed number of CB HSCs ≥0.0010% were associated with a significantly lower risk of IVH, RDS, and infections, with borderline statistical significance (p = 0.092) for anemia.
Models with gestational nutritional indicators as the main predictor have also been adjusted for maternal HIV status.
In order to explore the role of the MAPK and apoptotic pathways in the development and resolution of the cardiomyopathy seen in infants of diabetic mothers, we developed two rat models of gestational hyperglycemia – one with very poorly controlled glucose with maternal ketosis and a second model with moderately well controlled glucose levels and no maternal ketosis.
Therefore, we addressed in murine models whether gestational treatment with mycobacterial antigens could induce better immune responses in the postnatal life.
For both the unadjusted model with only gestational age in the mean regression, and the adjusted model with covariates in the mean regression, the ACE model with additive genetic (A), common environmental (C), and unique environmental (E) variances fitted best.
Related(19)
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