Exact(1)
Using a lumbar 5 (L5) spinal nerve ligation (SNL) pain model, we testified our hypothesis that SDH neurons and astrocytes reciprocally regulate each other to maintain the persistent neuropathic pain states.
Similar(59)
By comparing with evolutionary and dinucleotide constrained background models we testified that the discovered signals are statistically significant and are not likely to be attributed to factors such as mutation bias of amino acid composition in the investigated sequences.
We testified the sensitivity with a normal liver cell line LO2.
Here, we testified that NPC1L1−/− mice were resistant to colitis-associated tumorigenesis.
So we testified the cytoskeleton changes by fluorescence microscopy and Western blotting.
Furthermore, we testified that p53 over expression in EML cells causes down regulation of Malat1.
So, we testify properties (i)–(iv).
We testify the proposed approach on two real-world microarray datasets.
We testify the proposed approach on two real-world microarray datasets of different statistical characteristics.
To testify the dust model, we first fit the standard extinction curve of RV = 3.1.
The model was testified to its feasibility and rationality by a practical illustrative example.
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