Using a simple bet-hedging model, we show analytically and computationally that PCD can be adaptive.
Moreover within the framework of the neoclassical model we show the standard prescription regarding a corrective tax to be correct.
In the rewire-to-random model, we show that a positive fraction of both opinions survive with high probability.
Using a computational model we show that the two theories are not mutually exclusive.
Using a simple network model, we show analytically the effectiveness of the estimation algorithm.
Using a rate-and-state friction model, we show that effective normal stress is temporally variable on the fault, and support this using seismic observations.
Using this mouse model, we show here the dynamics of hepatic infiltration and exhaustion of NK and CD8+ T cells during acute HCV infection.
Using this model, we show that regulatory T cells (T reg) cells) and invariant NKT cells (iNKT cells) perceived stronger TCR signals than conventional T cells during development.
Employing a chemotaxis pathway model, we show that the gain of the pathway at the level of the response regulator CheY increases with overall chemotaxis protein abundances.
In support of this model, we show that mutations in the fly gene eiger, a TNF homolog, delay the lethality of Salmonella infection.
By using a humanized sickle cell anemia mouse model, we show that mice can be rescued after transplantation with hematopoietic progenitors obtained in vitro from autologous iPS cells.
