Consistent with this model, we observed that addition of PexA to an attC and attP integration reaction blocks Int-mediated recombination in vitro (Fig. 4d).
By using equivalent electrical model, we observed the dynamic processes of the discharge.
To judge the discrimination ability of the D-CNN model, we observed the distributions of the different neurons' excitations.
In the Snord116 deletion mouse model, we observed normal locomotor activity and excellent wire-hanging ability.
In contrast, using the AOM/Il10−/− model, we observed a strong correlation between inflammation score and tumor multiplicity.
Also in the Guinea pig TB animal model we observed a strong correlation between vaccine efficacy and antigen dose.
As others have seen in the B16-F10 model, we observed no curative effect of the antibodies without cellular vaccination (Figure S1) [14], [14].
In the working heart model we observed a similar reduction of contractility under ischemic conditions in CTGF-TG and WT hearts.
In the oligodendroglial lineage model we observed that 1 µmol melatonin exposure was associated with an enhancement of oligodendroglial maturation to myelinating oligodendrocytes (Figures 8A, 8B).
In a 6 weeks old CD1 septicemic mouse model, we observed that PilB was dispensable for bacterial virulence in both genetic backgrounds.
Using this model, we observed syndecan-1 signal persisted in the Mmp7−/− epithelium after injury but was diminished in WT airway epithelium (Figure 1B).
