Based on this new model, we demonstrate various diffusion strategies for firms of different size.
Using a simple intertemporal optimization model, we demonstrate why this information is not sufficient to design emission reduction strategies.
As an application of our proposed model, we demonstrate how a stress field in an electrodic domain may significantly affect the evolution of an anodic dissolution front.
Through numerical examples for the Marmousi model, we demonstrate that our inversion algorithms can reproduce the subsurface velocity structure without estimating source signature.
Using our model we demonstrate that under strategy 1 complete chondrogenesis will be induced if the initial proportion of chondrocytes exceeds a critical value.
Screening descriptors calculated through a semi-empirical model, we demonstrate how an informatics-based analysis can be used to address this issue with no prior assumptions.
Using a Drosophila model, we demonstrate that IRs act in combinations of up to three subunits, comprising individual odor-specific receptors and one or two broadly expressed coreceptors.
Using a mouse model, we demonstrate robust phenotypic reversal, as activation of MeCP2 expression leads to striking loss of advanced neurological symptoms in both immature and mature adult animals.
Although ours is not a lung injury model, we demonstrate that prone repositioning helps even the regional distributions of aeration and ventilation when compared to supine or lateral decubitus positions, even in this setting of highly unilateral mechanical abnormality.
Using a hyphal growth model we demonstrate here for the first time how to optimize bioincising by linking the microscopic growth behavior of P. vitreus with macroscopic system properties of the wood.
Using trio-interaction model, we demonstrate the frictional convergence feedback provides a mechanism that couples the Kelvin wave and Rossby wave together along with convective heating, and selects eastward propagation.
