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Using the GAL network as an experimental model, we characterized cross-species evolution of this network and how cellular aging affected the network activity.
In this work, using Caco-2 cell model, we characterized the cellular transport pathway of pegylated polyester nanoparticles and determined the effect of polymer architecture including PEG chain length and core material on its cellular interaction and transcellular transport.
In this enterocyte model, we characterized the partners of PrPc in cell cell junctions.
Using the Lewis lung carcinoma (LL2) implantation mouse model, we characterized the hematopoietic compartment in the tumor stroma.
Using a murine lung epithelial cell line, primary lung epithelial cells and an inhalational murine infection model, we characterized the types of innate immunity proteins and peptides produced upon B. pseudomallei infection.
Thanks to our in vivo model, we characterized for the first time, the host immunological environment allowing the maturation of Mikulicz cells.
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For this simple model, we characterize explicitly all possible equilibrium configurations.
Using our model, we characterize the compensating differential for employer-sponsored health insurance (ESHI) — the causal change in wages associated with gaining ESHI.
Using a thermodynamic model, we characterize the interplay between TF copy number and the demand for that TF.
By adopting an additive quantization noise model, we characterize optimal coder-decoder pairs and show how to allocate individual bit-rates.
For this model we characterize the joint probability generating function in terms of a system of partial differential equations; this system enables the evaluation of (transient as well as stationary) moments.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com