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In the basic (ACE) model, variation is assumed to arise from three sources: (1) additive genetic effects (a); (2) common environmental effects (c); and (3) unique environmental effects (e).
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In addition, some causes of model variation are much more prosaic than the causes considered by cognitive diagnostic models.
Every model variation was thus trained using 544 (16 samples × 34 variables) spectral data points, with a blind test set of 34 spectral data points.
We next asked if the exon model variation was due to a small number of genotypic changes (some with a large effect) or many changes (each with a small effect).
Because 3bej-1 was very restrictive, a model variation was created that only contained the chemical features on the steroidal core and the neighboring atoms.
Second, in the so-called Speaker Design model, stylistic variation is seen as a process of identity building.
Even under the neutrality conditions of our model, any variation is tolerated so long as the expression dynamics are unaffected.
Conversely, it is unknown how well the Bayes B method will perform when the true model of variation is more "infinitesimal".
In our model, the variation is set by a persistence length, which specifies how much a tube would elongate before losing a significant component of its original direction.
Like the sexual antagonism theory, the coadaptation theory employs a standard assumption of quantitative genetic models: that variation is available and persistent.
We tested for the heritability of the phenotype by comparing likelihood between the reduced model, where total variation is due to environmental variation only, and the full model where total variation is composed of environmental and genetic effects estimated from the genetic relationship coefficient of each pair of individuals.
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