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If the items and persons fit the model, these interaction statistics would have a mean of approximately zero and a standard deviation (SD) of one.
Treating combinations of components as clinically meaningful units encompasses the complex intersections, synergies and conflicts between components, though it misses the opportunity to specifically model these interaction effects.
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Circuits that model these interactions are also presented in this paper.
For nanopeapods with linearly arranged nested fullerenes, equivalent pressure distribution is proposed to model these interactions.
Cyber-physics system (CPS) engineering methods could provide valuable insights to help model these interactions and optimize the design of such systems.
We also discuss gene environment interactions, a major challenge in complex disease genetics, and we present examples of cell-based approaches that model these interactions in vitro under controlled conditions.
In our model these interactions are schematized via the sequence-matching rule for the sharing of information between the cognates/cognate response elements.
In order to model these interactions, functional data on each individual factor will be required.
In all but the neurological model these interactions were both positive and negative.
According to our model, these interactions are supposed to have two effects.
The complex nature of this information motivates the search for analytical tools that can model these interactions to examine associations between gene interactions and cancer.
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Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com