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To model the virus's spread in people, they housed ferrets inoculated with the reassortants in cages adjacent to healthy ferrets so the animals could pass on virus through the air.

According to this model, the virus would evolve over a neutral fitness landscape and many of the mutations would be synonymous in nature.

In the two target cell model the virus

The Evolutionary (EV) engine uses derived evolutionary features to model the virus's expected escape path from drug therapy.

Inasmuch as model (1) does not explicitly model the virus dynamics, we can incorporate the effect of virus heterogeneity into the infected cell population.

In another human experimental norovirus infection model, the virus was detected in stool samples for a median of 4 weeks and for up to 8 weeks after virus inoculation [ 27].

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They make use of partial differential equations (PDEs) to model the virus-tumor system, because PDEs provide efficient information on the spatial and reactive mechanisms affecting the wave propagating fronts and PDEs can be used to compute their speeds.

The generated viruses showed preferential replication in tumor cells and in a subcutaneous HT-29 colon cancer xenograft model the viruses demonstrated significant oncolytic activity comparable with dl327.

To explore the model behaviour, the virus spread was simulated over 10 years after the introduction of a near-calving PI heifer into a susceptible 38 cow herd.

The model initiates the virus by infecting an AP at random to act as a seed and then calculates how many days would be required to either infect or blacklist all APs in the area.

Ralph Baric of the University of North Carolina, Chapel Hill, and others argued that their attempts to adapt the MERS virus to mice in order to develop an animal model for the virus are crucial for vaccine and drug studies.

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