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Exact(6)

The photometric assays were accomplished, employing as a model, the compound formed reacting iron (III) with thiocyanate in acidic medium.

The receptance theory is introduced to model the compound system consisting of the host beam and the attached beam-based DVA.

In the cell model, the compound also provides a higher antioxidant capacity than α-tocopherol and lycopene, alone or in combination, suggesting the possibility of an oxidative intramolecular cooperation.

In the steroid receptor model, the compound enters the cell cytoplasm where it acts as a specific ligand, binding selectively to a high affinity receptor protein.

Although encoding cell lines with cell line kernels may be sufficient to model the compound pGI50 values from the NCI60 panel, in our view this would not be the case on datasets comprising highly dissimilar cell lines.

Finally, docking of S20 with the compound-sensitive HA from A/Vietnam/1203/04 (H5N1) (PDB 2FK0) protein yielded a similar binding pocket within the HA2 loop structure, whereas attempts to model the compound with the S20-resistant HA from A/Aichi/2/68 (H3N2) (PDB 2YPG) yielded no predicted binding pockets within this region.

Similar(54)

By modeling the compound fragmentation as a tree, we make the implicit assumption that each fragment in the fragmentation spectrum is generated by a single fragmentation pathway.

An intra-dimensional shift (IDS) modeled the compound discrimination paradigm with, importantly, novel odors and media for each IDS.

In rodent models, the compound inhibits the development of cancers of the skin, duodenum, tongue, colon, mammary, and prostate glands [ 22, 23].

For these data sets, performance progression between the label removal model and the compound removal model was very similar.

We added random intercepts for each model and the compound symmetry correlation structure was considered.

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