Animal model studies indicate that hypercholesterolemia increases neutrophil counts and that neutrophils infiltrate arteries in the early stages of atherosclerosis[35].
Model studies indicate that this configuration favors the targeted delivery of NaCl to loop bends, where a favorable gradient, sustained by urea absorption from collecting ducts, promotes NaCl absorption.
Model studies indicate that nonlinear interactions contribute to the TDT preferably at low and midlatitudes (Smith and Ortland 2001; Huang et al. 2007).
In vitro and animal model studies indicate that high and disseminated viral replication is an important factor in disease pathogenesis [4] [7].
The results were consistent in the genetic model studies, indicating that our results are statistically robust.
Autopsy and animal model studies indicate severe inflammation of the vessel walls (vasculitis) as a key etiology [ 3, 4].
Mouse model studies indicate that both DNA-PKcs deficiency and WRN deficiency synergize with telomerase loss and shortened telomeres to accelerate the onset of aging related phenotypes.
The delivery time was based on prior animal model studies indicating the efficacy of short duration of IACFM applications [ 16- 18].
Mouse model study indicated that mutation of RNF8 will compromise normal spermatogenesis, as well as showing increased sensitivity to ionizing radiation.
