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Animal model studies indicate that hypercholesterolemia increases neutrophil counts and that neutrophils infiltrate arteries in the early stages of atherosclerosis[35].
Model studies indicate that nonlinear interactions contribute to the TDT preferably at low and midlatitudes (Smith and Ortland 2001; Huang et al. 2007).
In vitro and animal model studies indicate that high and disseminated viral replication is an important factor in disease pathogenesis [4] [7].
Autopsy and animal model studies indicate severe inflammation of the vessel walls (vasculitis) as a key etiology [ 3, 4].
Mouse model studies indicate that both DNA-PKcs deficiency and WRN deficiency synergize with telomerase loss and shortened telomeres to accelerate the onset of aging related phenotypes.
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Additionally, transgenic animal model studies indicated that tumourigenesis does not require gene amplification [ 18].
The results were consistent in the genetic model studies, indicating that our results are statistically robust.
The delivery time was based on prior animal model studies indicating the efficacy of short duration of IACFM applications [ 16- 18].
Laboratory and animal model studies indicated that zinc and iron compete for absorption in the intestinal lumen as they have similar physicochemical properties [ 33- 35].
Our molecular modeling studies indicate that the other antagonists bind in a similar fashion.
Although the effect of flow was not studied in our patients, the model study indicates that overestimation should be higher at low flow.
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