Sentence examples for model studies demonstrated from inspiring English sources

Previous mouse model studies demonstrated immunization with r-GV expressing selected exogenous sequences elicited a prolonged immune response.

Previous DS animal model studies demonstrated that ETS2 had a tumor suppression role in intestinal tumor oncogenesis (41).

Several works in epidemiology and animal model studies demonstrated that compounds which possess antioxidant or anti-inflammatory properties can inhibit carcinogenesis [ 6, 7, 8, 9].

Animal model studies demonstrated that loss of TACSTD2 did not alter the incidence and natural history of papilloma formation, but enhance malignant phenotype of skin cancers through promoting EMT.

We review evidence from clinical data sets and mouse model studies demonstrating enhanced inflammation and altered tumor microenvironment (TME) upon TS inactivation.

- Animal model studies demonstrate that increased bile acid levels in the colon select against the Bacteroidetes and Actinobacteria and favor the Firmicutes including bile acid 7α-dehydroxylating bacteria in vivo.

While there are some cell model studies demonstrating activation of IGF-IR by overexpression of receptor or intracellular tyrosine kinases (Kozma and Weber, 1990), activation of IGF-IR requires binding to ligand in most physiologic settings.

Our results are consistent with findings derived from animal model studies demonstrating that the use of a SRL dosage similar to that prescribed in KT was associated with regression of pressure-induced cardiac hypertrophy by means of antiproliferative mechanisms [ 3- 5].

The molecular modeling studies demonstrated that compounds matched the pharmacophore well and had good predicted physicochemical properties and estimated IC50 values.

Recent numerical modelling studies demonstrated how pre-existing (geologically older) fault geometries within a rock volume, strongly control both the distribution of strain and fluid flow patterns during extensional fault reactivation.

Modelling studies demonstrated that monomeric α-syn can rapidly penetrate the lipid bilayer and incorporate additional monomers to form pore-like structures that fully perforate the lipid bilayer [ 49].