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Two- and three-dimensional model simulations revealed the heterogeneous current distribution over the planar anode surface for younger and patchy biofilms, but becoming uniform in older and more homogeneous biofilms.
Model simulations revealed that for systems employing high ratios of TEMPO KPS, the consumption of TEMPO by polymer radicals derived from KPS decomposition and styrene thermal initiation (using the accepted literature kinetic rates) is not sufficient to lower TEMPO concentrations to levels where polymer growth can occur.
Patients with schizophrenia were characterized by the decreased gamma band power and increased beta band synchronization relative to healthy controls, and network model simulations revealed that such abnormalities resulted from deficits in the CLGF circuit.
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Model simulations reveal non-intuitive effects of drug combinations [17].
Model simulations reveal that at higher values of solid conductivity the catalyst does not exhibit hot spot both in time and space, but needs higher inlet gas temperatures to reach complete methane conversion.
Model simulations reveal that increasing the inlet flow rate and outlet fluid pressure increases oxygen delivery to the cell layer and, therefore, the growth rate of cells that are tolerant to high shear stresses, but may be detrimental for shear-sensitive cells.
The model simulations reveal that just a spatial low-pass filter combined with a presynaptic shunting inhibition (and implicitly assumed linear transmission characteristics at all synapses) are sufficient to produce a preference for small objects (fig. 11).
Model simulations reveal that vascular stiffening increases sympathetic tone and shifts the effective baroreflex response to an increased pressure baseline.
These model simulations reveal that the activated AP-1 activity response arises from a dynamic balance of constituent AP-1 dimer forms.
In particular, our model simulations reveal that the experimentally observed AP-1 sensitivities to pathway inhibitors should be interpreted with caution as these effects are dynamic and yield contrasting results based on the time point of interest.
As shown in Figure 4A, model simulations reveal that the activation of AT1R with FRK inhibition eliminates the AP-1 response during the first 20 minutes, while restoring the AP-1 response to the uninhibited levels at late time points.
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CEO of Professional Science Editing for Scientists @ prosciediting.com