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The lateral redistribution given by the model revealed from 5%to25%5% less water on the crests compared to in the valleys for metamorphic, sand and sedimentary bedrocks.
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Simulation from the model revealed that an increase in permeate flux from 10 to 30 led2h led to an increase in the rejection of low molecular weight N-nitrosamines such as N-nitrosodimethylamine (NDMA) (from 31%to5454%), which was validated by experimental results.
Further analysis with the model revealed that the reaction from ferulic acid to feruloyl-CoA, which is catalyzed by 4CL, is the bottleneck.
Meanwhile, the derived contour maps from 3D-QSAR model revealed the significant structural features (steric and electronic effects) required for improving CETP inhibitory activity.
Graphical inspection of autocorrelelogram from this model revealed residual correlation between the number of laboratory reports on a given day and the 21 days previous.
Analysis of tumors from this model revealed that they exhibited strong bone resorption, induction of osteoclasts and intracranial penetration at the tumor bone (TB -interface.
The results from the structural model revealed that the proposed relationships among study variables are positively and significantly associated.
The information extracted from the QSAR model revealed that the major driving forces for radical scavenging activity are hydrogen bond donation and polarity.
A comparison between experimental results and results predicted from the mathematical model revealed that the model can describe the essential effects of decomposition kinetics.
Our results from the mouse model revealed that spatacsin disruption intervenes axonal stability and SV transport.
The classic FRS model (figures in parentheses are from the modified model) revealed that 20.5% (21.8%) and 38.46% (38.9%) of respondents were at high and moderate risk of CVD.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com