The observation that Tpl2 deficiency is protective in our VILI model prompted us to examine the potential therapeutic effect of pharmacologic inhibition of Tpl2 in high VT-induced lung injury.
The success of this model prompted us to use it to test the efficacy of the protocol we have established for infection detection using molecular biology methodology ([6],[8]; Mariani et al. 1996b; [7],[1996b1996b
The data obtained in the human tumor-SCID mouse model prompted us to investigate whether the Exotest allowed the detection and characterization of exosomes purified from human plasma.
This model prompted us to perform a microarray screen to search for nuclear-enriched non-coding RNAs, using XIST's properties as a guide.
This feature of the Simplified cAMP Model prompted us to develop the Complete Model.
The positive effects of ibuprofen in the transgenic model prompted study of the compound in peptide-injected rat brain.
The presence of hornerin in each stage of the MCF10A breast cancer progression model prompted us to investigate hornerin expression in correlation to breast cancer subtype.
SEMNET discussion of this model prompted Hayduk to investigate whether some unrecognized measurement feature was producing the significant, even if seemingly slight, ill fit.
The finding from the EAE model prompted us to test whether STAT5-deficient T cells also lacked pathogenicity in other T cell-mediated autoimmune diseases.
These in vivo studies in the mouse CCI model prompted us to examine directly whether Akt and mTOR mediate programmed necrosis in neuronal cells.
