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Sivick, K. E., Schaller, M. A., Smith, S. N. & Mobley, H. L. The innate immune response to uropathogenic Escherichia coli involves IL-17A in a murine model of urinary tract infection.
The objective of the study was to investigate bone strength at four different skeletal sites in a chronic animal model of urinary diversion.
These compounds lack intrinsic antibacterial activity in vitro and were able to limit a bacterial infection in a mouse model of urinary tract infection.
The fnr mutant of E. coli results in severe defects in adherence to and the invasion of bladder and kidney epithelial cells and was highly attenuated in a mouse model of urinary tract infections (Barbieri et al. 2014).
In order to begin to define the role of the host in disease, we developed a rodent model of urinary tract infection (UTI) using Fischer 344 (F344) rats.
Our multiple regression model of urinary 3HPMA among tobacco smokers (Table 3), adjusted for sex, age, and race/ethnicity, showed that serum cotinine was positively associated with 3HPMA levels in urine (p < 0.0001).
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Animal models of urinary incontinence and erectile dysfunction following radical prostatectomy (RP) are lacking.
Murine models of urinary tract infection (UTI) have provided substantial data identifying uropathogenic E. coli (UPEC) virulence factors and assessing their expression in vivo.
Cefoxitin and temocillin have proven efficacy in murine models of urinary sepsis [50, 51], yet clinical data are still pending [52].
Table 4 presents results of final linear regression models of urinary markers.
The base models of urinary and hair mercury were combined with main effect and interaction terms between the respective exposure predictors and dummy variables of each SNP.
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