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An alternative approach would be to examine the inflammatory cytokine response to docetaxel administration alone in a mouse model of sickness behavior.
Importantly, this model of sickness behavior does not contradict the development of hypersensitivity to noningestive stimuli through classical conditioning.
There is also some evidence suggesting that NO modulates the function of hypothalamic arcuate nucleus (ARC) in an animal model of sickness anorexia induced by bacterial endotoxin lipopolysaccharide (LPS) injection [ 17]. iNOS-dependent NO formation inhibits orexigenic NPY-containing arcuate neurons that are excited by ghrelin.
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In addition, we stratified the regression models of sickness and disability following dropout across the quintiles of the propensity score for high school dropout.
Consequently, analytical approaches to modeling of sickness absence duration should take into account both event dependence and heterogeneity to avoid obtaining biased estimates of the parameters of interest [ 11, 12].
The combination of theoretical models were the empowerment model, theories of planned behaviour and personal models of sickness [ 37], the empowerment model and the discovery learning theory [ 38], the social cognitive theory and the social ecological theory [ 50], the self-efficacy and self-management theories [ 55] and the cognitive theory and operant reinforcement theory [ 56].
Animal models of serum sickness, a circulating immune complex disease, have been shown to develop cellular infiltrates in the uvea [7].
First, we derive a mathematical model of the motion sickness incidence caused by the head movement in 3D space based on subjective vertical conflict.
Without an experimental model of equine grass sickness (EGS), a randomised controlled field trial (RCT) represents the only method of evaluating the efficacy of Clostridium botulinum type C vaccination in preventing naturally occurring disease.
The present study, using an experimental rat model of human sleeping sickness (HAT), was designed to evaluate the changes occurring in NO production throughout the time-course of T. b. brucei infection.
Table 2, fully adjusted model, shows higher odds of sickness absence for those who received the treatment combinations drug treatment, psychotherapy and physiotherapy (OR 1.67, 95% CI 1.25 to 2.25), compared with those who did not.
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