Exact(4)
The aim of this study was to develop a conceptual model of saline intrusion within coastal karst aquifers by analyzing Rainfall Discharge Salinity data and to assess freshwater resources using a lumped modeling approach.
Hyperinflation following repeated OES can deteriorate arterial oxygenation and lung injury in a rabbit model of saline lavage induced lung injury undergoing mechanical ventilation.
In an ex vivo lung model of saline lavage, the use of high PEEP combined with a low TV was demonstrated to reduce histological VILI [ 5] and to reduce the resulting inflammatory reaction [ 6].
Also, a recent model of saline lavage lung injury found that high tidal volume/low PEEP ventilation resulted in lung injury in the non-dependent lung regions in supine rats, suggesting atelectasis in the dependent lung zones shifts stretch-induced injury to the non-dependent lung and argues against repetitive collapse and expansion as a cause of VILI [ 17].
Similar(56)
Van der Kloot and colleagues [ 17] studied the effects of RMs on gas exchange and lung volumes in three experimental models of ALI: saline lavage, oleic acid, and pneumonia.
Based on this hypothesis, Kloot et al. [ 17] investigated the effects of recruitment maneuvers on gas exchange and lung volumes in three experimental models of ALI: Saline lavage or surfactant depletion, oleic acid, and pneumonia, and observed improvement in oxygenation only in ALI induced by surfactant depletion.
Another popular model - the Gadanis model for saline water [5] - we use to determine a complex permittivity of the saline water.
Kostic and colleagues [ 20] evaluated the use of respiratory system reactance to identify the minimal PEEP level required to maintain lung recruitment in a porcine model of ARDS induced by saline lavage.
Because of complex and heterogenous mechanisms of human ARDS, we used a rat model of ARDS induced by saline lavage and mechanical ventilation to perform miRNA and mRNA microarray analyses simultaneously, aiming to identify miRNA-mRNA interactions and to understand the impact of these interactions on the pathogenesis of ARDS.
The hsryfm 1301 group and hsryfm 1301-f greceivedeivedailyly 2 mL (10 CFU/mL) of L. rhamnosus hsryfm 1301-containing skimilklk suspension and L. rhamnosus hsryfm 1301-containing skim fermented milk, respectively, which was administered intragastrically for 28 d, after the hyperlipidemic rat model was established The control and model groups received an equivalent volume of saline.
If we assume a Hashin-Shtrikman model using saline fluids of 0.1 Ωm, a porosity of 1.5% to 5% would be required to explain the bulk resistivity of 1 to 10 Ωm.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com