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This model is an extension of the lumped model of methyl butanoate developed and discussed in the first part of this work [1].
To illustrate the practical utility of the input sequences considered here, second-order Volterra and Volterra Laguerre models are developed that approximate the dynamics of a first-principles model of methyl methacrylate polymerization.
Thus, the lumped kinetic model of methyl butanoate and methyl decanoate is also quite simply applied to simulating the combustion behavior of intermediate methyl esters, by using the lever rule between the two reference components.
The three-dimensional (3D) model of methyl jasmonate esterase (MJE), which is only responsible for methyl jasmonate (MeJA -cleaving activity, is constructed based on the crystal structure of salicylic acid-binding protein 2 (SABP2, PDB code 1XKL) by using InsightII/HoMeJA -cleaving activityher refised using unrestrained dynamiconstructedons.
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Thus, DNA methylation is upstream of histone modification in this model of methyl-DNA binding proteins and histone modification.
In a rat model of methyl-deficient or lipotrope-deficient diet-induced HCC, global hypomethylation has been found to be associated with tumor development [ 68].
The data were modeled using two recently developed kinetic models of methyl formate oxidation, and significant disagreements were realized at all pressures especially under fuel-rich conditions.
The contribution of the phosphor to the photocatalytic model reaction of methyl orange dye decomposition in aqueous solution was experimentally verified under power-saving illumination conditions.
A chemical kinetic submechanism proposed here is an essential prerequisite to model autoignition of methyl esters at low temperature region (less than ∼900 K), where reactions of methyl-ester peroxy radicals (ROO) and hydroperoxy methyl-ester radicals (QOOH) are crucial and relatively unexplored.
From the model reaction of methyl propiolate (1) with benzylmercaptan in the presence of tri-n-butylphosphine catalyst, the corresponding dithioacetal was obtained in high yield when the reaction was carried out under higher concentrations of the catalyst and the reactants.
Previous studies have demonstrated that miR-155 plays an important role in hepatic lipid metabolism, has a protective role against HF diet-induced non alcoholic liver steatosis [ 33], and was found to be up-regulated in NASH models of methyl-deficient diet, in HCC induced by choline-deficient and amino acid-defined diet, and in primary human HCC [ 34, 35].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com